Preliminary studies suggest conolidine could inhibit unique ion channels, minimizing neuronal excitability and limiting discomfort signals. This mechanism is especially related in neuropathic pain, the place abnormal signaling causes persistent discomfort. Also, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to agony perception. Computational docking ex... https://philipl849gps3.wikiconversation.com/user